​LA MALADIE DE LYME ET LES MALADIES CHRONIQUES Dietrich Klinghardt

Dietrich Klinghardt (contribution 2008) sur DVD sous www.ink.ag et www.steinhardtverlag.de 


​Le Dr Klinghardt montre des images au microscope de nombreuses borrélies qui entourent un globule blanc, et le terrassent. ​Normalement les microbes fuient devant les cellules tueuses du système immunitaire, cependant les borrélies attaquent, endommagent et tuent ces dernières, ou bien ils les pénètrent et vivent intracellulaires. ​Ce qui est triste c'est que l'école de médecine part du fait que lorsque le système immunitaire ne produit pas d'anticorps, le patient n'a pas de borréliose. D'autre part les anticorps doivent être trouvés dans le sang où les borrélies ne se trouvent pas, elles vivent de préférence dans les tissus et sont destructrices des cellules des tissus. ​C'est un non sens que le standard du diagnostic soit ce test d'anticorps. ​Etablir la présence de borrélies de la plus facile et meilleure façon est le microscope. Tout du moins dans les stades ultérieurs, chroniques, quand le système immunitaire est au plus bas à cause de l'activité des agents pathogènes et effondré, la preuve doit se faire au microscope, pas avec la méthode indirecte. ​On n'examine pas assez au microscope, c'est devenu presqu'étranger à la médecine, on gagne moins d'argent qu'avec les examens de laboratoire. Nous devrions beaucoup plus regarder au microscope pour voir directement les agents pathogènes. Ils sont présents dans le collagène, dans les tissus, les nerfs, les vaisseaux, les fascias, la peau, les muqueuse, et même dans les cellules cancéreuses. ​Quand on traite aux antibiotiques au minimum 3 mois, les scientifiques les plus rigoureux ainsi que moi-même signifions que dans les premiers stades, il faut plutôt traiter 1 an1/2, car après 1 an1/2 les kystes disparaissent. ​Les kystes se formeraient pendant le traitement antibiotique pour protéger les agents pathogènes et les rendre inaccessibles aux médicaments. Mais ces kystes ont un temps déterminé de survie. C'est pourquoi tant de patients rechutent après un temps trop court de traitement. ​Dans un stade tardif, il est important de combiner les antibiotiques.et de toujours penser aux co-infections. ​Au début, avec la bartonella, associer quinolone et tétracyclyne, au 2ème mois avec babesia zithromax plus mepron un antimalarique., au 3 ème mois des perfusions de rocéphine avec du métronidazole ou tinidazol(fasigyne). C'est un programme de base, si vous avez reçu un traitement trop court et non combiné, c'est comme si vous n'aviez pas été traité.
2 ​Les noyaux de la bactérie se protègent les uns les autres et construisent avec d'autres noyaux des couches de bio-film principalement dans l'intestin, le cerveau, le système nerveux, les fascias, les dents. ​Ces bio-films sont chargés de toxine de telle sorte que le système immunitaire ne peut y pénétrer et que ce sont les toxines qui occasionnent les symptômes de la borréliose, et pas les noyaux de la borrélie directement. Cela vaudrait pour les toxines qui occasionnent, réactions et fausses réactions du système immunitaire.Dans la guérison la génétique tient un rôle essentiel, ​Il y a peu de patients atteints de sclérose en plaque pour lesquels on ne retrouve pas de borrélies. L'enveloppe de la borrélie avec ses antigènes n'est pas différenciable de la myéline des nerfs, les spirochètes ressemblent aux cellules du système nerveux. ​Le système immunitaire a 2 possibilités, ou bien il laisse les deux en paix ou il attaque les spirochètes et les couches de myéline. Dans la sclérose en plaque, la myéline est attaquée car elle ressemble à l'enveloppe des spirochètes. Pourquoi donc ne pas combattre les bactéries dans la sclérose en plaque plutôt que d'administrer des médicaments ? ​Quelles sont les maladies qui sont typique d'une borreliose chronique non reconnue ou trop tard ou mal soignée ? 

 Les maladies neurologiques, et psychiatriques telles les polyneuropathies, polyradiculites, polymyalgies, myelopathie, paralysie, SLA, SEP, dépression, schizophrénie, démence … La neuroborreliose, ressemble à la syphilis. ​Chez presque tous les patients atteints d'Alzheimer on peut trouver des spirochètes dans le cerveau, l'herpès est souvent concomittant., et quand on traite précocément on peut stabiliser les malades du Parkinson ​Dans le problème de thyroïde, les problèmes auto-immuns comme le lupus la sclérodermie, la polyalgie ce serait identique.

ANOREXIE​ Par ailleurs, j'ai traité 8 jeunes filles dans les 5 dernières années par des thérapies antimicrobiennes. ​Beaucoup de malades de la borreliose ont un rythme cardiaque accéléré, la cardiomyopathie et l'arythmie sont fréquentes. ​L'épilepsie chez les enfants se trouve souvent associée avec la bartonella, les enfants ne devraient pas avoir de chats à la maison, ils sont porteurs d'agents pathogèness comme la toxoplasmose, la tularémie, la bartonellose et toutes sortes de virus. Sans virus d'oreillons, d'herpès, ou de borrelies, il n'y a pas de sclérose en plaque.
 Dans la SLA il y a présence de mycoplasme, dans le Parkinson, bartonella et babesia. Une des causes du cancer du sein est le virus d'Epstein-Barr. Les mycoplasmes et d'autres virus y participent.
 ​Une des causes les plus fréquentes de CFS (fatigue chronique ?) est le virus humain de l'herpès type 6 fabriqué par les militaires, les borrélies, Epstein Barr et les staphylocoques (coagulase négative) sont incriminés ainsi que la pollution environnementale, les amalgames dentaires au mercure, l'électro-pollution.

3 ​ ​Il existe des liens entre la borreliose et les amalgames. Des tâches rouges à proximité du ventre indiquent une babesiose. ​Seulement 24 % des malades de borreliose ont des problèmes d'articulation tels que l'arthrite. Il faut noter que le système immunitaire est affaibli par une une longue exposition aux agents pathogènes ou à des expositions répétées. ​ ​Dans le cas de la borréliose, le système immunitaire n'est pas véritablement affaibli, c'est seulement des parties du système qui le sont , qui sont désorientées et ineffectifs. Un bon système immunitaire vient à bout de tout. ​Chez seulement 25 % des patients survient un E.M. Je suppose que l'E.M est le signe d'une réinfection. Lors de la première piqure il y a rarement cet E.M ​Les cas « lourds » indiquent des réinfections et co-infections. ​A propos du test avec les AC, le test devient positif quand le malade est traité, par exemple après un traitement antibiotique la charge d'agents pathogènes est réduite et les lymphocytes peuvent réagir et produire des A.C. ​Après un contact avec la borrélie, une paire de minutes après, elles sont dans le système nerveux. C'est étonnant à quelle vitesses elles s'introduisent dans les tissus, elles sont plus rapides que le sang et les cellules immunitaires. En quelques secondes, les borrélies avec leurs mouvements en forme de vrille s'infiltrent de la tête à la vessie pas par le sang mais les tissus.Les borrelies peuvent être transmises d'être humain à être humain par relation sexuelle, une fois suffit, le sperme et les sécrétions sexuelles féminines sont souvent pleines de spirochètes. Dans 1/3 des flacons de sang de transfusion, il y a des borrélies (non contrôlés) les moustiques et les puces sont vecteurs. ​>                                                                                                                                                          Transmission au foetus : beaucoup d'enfants viennent au monde normalement et présentent plus tard de l'hyperactivité des difficultés d'apprentissage, certains deviennent autistes après vaccination, un enfant infecté ne supporte pas les vaccinations. Les problèmes de fausse couche peuvent y être liés. Les borrélies sont dans le lait non pasteurisé. ​Lors d'une piqûre de tique diverses autres bactéries peuvent être transportées, borrelies, babesia, (80% des borrélioses) ehrlichia, coxiella, bartonella, mycoplasme (difficiles à soigner par antibiotiques, après 1 an ½ d'antibiotiques, seuls 60 % sont éliminés, des huiles de plantes ozonisées, et du peroxyde de plantes seraient plus efficaces) ​Le meilleur test de dépistage des mycoplasmes est le CD57. ​100 – 300 normal ​60-100 borreliose ​moins de 60 borreliose et mycoplasmes ​90 % des spirochètes vivent dans le système digestif, 10 % dans les tissus où ils causent la majeure partie des problèmes par exemple dans les nerfs ou le cerveau.                                                                                                                                                                                                                                                        4 ​Les microbes disposent de beaucoup de mécanismes d'action, l'un des plus importants est de mettre sous silence la production d'hormones. Les bactéries telles les borrélies qui vivent avec nous et le veulent le plus longtemps possibles inhibent la production de l'hormone de croissance (les spirochètes et les chlamydia). ​Les polluants tels que le mercure et les infections réduisent la production de testotérone et les réactions immunitaires, ainsi que les champs magnétiques produits par les téléphones cellulaires et les appareils électro-ménagers;​ ​La mélatonine commande le système veille -sommeil, le processus de vieillissement, protège du cancer, fournit l'énergie et soutient le coeur, augmente l'activité du système immunitaire et est un antioxydant potentiel, protège le cerveau des dommages dûs aux métaux lourds. ​La mélatonine est la substance qui détoxifie le cerveau, les nerfs. La propolis montre des effets similaires à la mélatonine et traverse la barrière hémato-encéphalique sans problèmes. La mélatonine est l'antidote, des polluants environnementaux, des métaux lourds, et des biotoxines de bactéries, de virus et champignons. Il faut éviter les champs électromagnétiques pour permettre à la mélatonine d'être produite en quantité suffisante. ​Ces combinaisons fatales aboutissent à l'augmentation rapide des maladies neurologiques surtout chez les enfants qui ont peu de protection au niveau du cerveau et les personnes âgées chez qui les maladie neurologiques progressent extraordinairement; ​Si par exemple on fait pousser des champignons sous une cage de Faraday on peut mesurer les mykotoxines qui se détachent et combien de toxines sont libérées et combien elles sont puissantes. ​Ensuite si l'on enlève ce couvercle et qu'on expose cette culture à l'influence d'un téléphone cellulaire, l'activité de cette culture augmente 600 fois, cela veut dire qu'il y a 600 fois plus de toxines libérées. Les bactéries réagissent au stress en libérant des toxines. Les bactéries communiquent avec nous au travers de la maladie et nous disent : depuis que vous avez ces téléphones nous n'allons pas bien et vous le faisons savoir. La pollution électrique mène à ce que les hormones qui font grossir augmentent et celles qui font perdre du poids diminuent. ​La pollution électrique et le mauvais sommeil copntribuent de manière importante à ce que les neurotoxines libérées par les bactéries nous fassent grossir car les tissus emmagasinent ces poisons et de cette façon protègent le système nerveux. ​Souvent être gros protège contre les toxines. ​Les maladies virales existent avec en arrière-plan un appauvrissement des méthylgroupes et une intoxication aux métaux lourds;
                                                                                                                                                                        PLUM ISLAND : Le virologue allemand Erich Taub dirigeait le laboratoire américain conçu pour la guerre biologique combina les borrélies avec des virus agressifs. ​Au début des années 1950, ill revint à Tübingen, le laboratoire dépérit, les militaires n'y avaient plus d'intérêt, la guerre de Corée était finie, le gouvernement américain était en faillite.  
                                                                                                                                                                                 5 ​Il y a des photos du laboratoire détruit avec à ses abords des chevreuils broutant qui partirent lors des marées et dispersèrent les agents pathogènes. ​Plum Island est situé près de la côte du Connecticut près de New York au milieu de la ligne de migration, ainsi beaucoup d'oiseaux atterrirent de l'Atlantique et s'en allèrent, emportant les bactéries. ​La vigne comporte depuis les 40 dernières années les bactéries avec mycoplasmes, le virus de l'herpès, le CMV combiné et nous supposons que c'est cette avalanche de combinaisons qui nous arrive dessus.                                                                                                                                                                             ​ ​ Le soin des infections chroniques comme la borréliose comprend 4 phases : ​1 – DEPOLLUTION ​(métaux lourds, biotoxines, pollution environnementale) ​ Les toxines comme consolidation des bactéries les rendent inaccessibles aux antibiotiques, aux herbes; contre tous les moyens qu'on met en oeuvre pour les détruire. ​ Premier pas, détruire cet édifice, nous avons un protocole avec des moyens naturels et puis de l'E.D.T.A, en perfusion, DMPA, DMSA, DMPE, un nouveau moyen OSR, la colestyramine fonctionne seulement chez 8 % des patients avec la borreliose. ​ ​Pour détoxifier ail séché réfrigéré et la coriandre,, des phospholipides, CVE, CGF, de la chlorella nanorisé pour le cerveau, l'hydrothérapie du colon, le drainage lymphatique, le sauna, le hamman, bains de pied à température progressive. ​ ​ Utiliser la mélatonine pour dépolluer des biotoxines, pas le glutathion, l'acide alpha lipon …? ​ Le téléphone cellulaire rend la barrière hémato-encéphalique perméable aux poisons. Toute bonne thérapie devrait commencer par l'amélioration du sommeil.                                                                            ​2 AMELIORATION DU METABOLISME ET DE LA PHYSIOLOGIE; ​ Normalisation des valeurs PH alimentation, bio, diète, vitamines, minéraux, acides aminés en concentration élevée suivant la situation, traiter tous les déficits. ​ Le magnésium oral nourrit les spirochètes, le fer nourrit les babésias. ​ Pas d'amalgame, pas de dents métallique, le moins de pollution électrique, vivre sainement, avoir une activité physique modéreée dans la nature, oxygénation,soutien du système immunitaire. ​
3 THERAPIE ANTI-MICROBIENNE ​ Combinaison d'herbes et de plantes énergisées rizole ? ​ Combat contre les bio-films avec allicin et natrium EDTA, terre qui soigne à jeun. Silicium, agents protecteurs :ail séché, congelé, ail des ours, chlorella (contre CMV, listéria) oméga 3 natrium chlorit (MMS), gamma et zet rizole 'ozonisation de plantes et huiles). ​ Combat contre les bio-films avec allicin et natrium EDTA, terre qui soigne à jeun. Silicium, agents protecteurs :ail séché, congelé, ail des ours, chlorella (contre CMV, listéria) oméga 3 natrium chlorit (MMS), gamma et zet rizole 'ozonisation de plantes et huiles). izole (ozonisation de plantes et huiles)
 4 PROTECTION DU SYSTEME IMMUNITAIRE ET IMMUNOMODULATION ​ Dans les infections chroniques et surcharges de toxiques, le système immunitaire est détourné, trompé, inhibé, les principaux symptômes reposent sur l'immunité, peu ou pas sur les agents pathogènes. ​ Il faudrait rétablir l'équilibre entre TH1, TH2, TH3, réduire les cytokines inflammatoires et optimiser les activités des N.K cellules. ​ Si possible utiliser les mesures mentionnées ci dessus.. ​ et en plus : psychothérapie, méditation, plantes énergisées, thérapie ENDERLEIN (cf document joint ENDERLEIN zoologiste et bactériologiste allemand), thérapies à photons, oxygène, antiviraux, antimycosiques, antiparasitaires.

nouveau cas de borreliose à VIRGINIA BEACH

New tick disease found in Hampton Roads

How to spot Tidewater Spotted Fever

Updated: Tuesday, 31 Jul 2012, 5:59 AM EDT
Published : Monday, 30 Jul 2012, 7:58 PM EDT
VIRGINIA BEACH, Va. (WAVY) - Scientists say nearly half of Gulf Coast ticks in Hampton Roads are carrying a new disease.
Jessica Crosley, 13, is one of only 15 reported cases of the disease in the U.S. She says she was in excruciating pain and experienced chills and a fever during 100-degree temperatures.
"I would scream it was so bad," Crosley said. "I've never been in any pain like that. I couldn't walk. Anything I ate I threw up."
Crosley told her mother she found a tiny black bug while showering.
"I had never seen a tick before in my life, so I just kind of ripped it off and it was like a lady bug" Crosley said.
Crosley and her mother found a white spot that over the days turned into a large black lesion.
"It looks a lot like a disease some are familiar with called Rocky Mountain Spotted Fever,"Infectious disease expert Dr. Randall Fisher at Children's Hospital of the King's Daughters said.
Crosley is only the second person ever to be seen with it at CHKD.
"The difference is where the tick bites, you end up getting a little black spot of skin that eventually breaks away and leaves a shallow ulcer," Fisher said.
The disease was first discovered here in Tidewater and is carried by the Gulf Coast tick. Scientists at Old Dominion University discovered nearly half of the Gulf Coast ticks in Hampton Roads carry it and issued a report to the government in 2011. The Center for Disease Control now lists it as an emerging disease.
"The good news about Tidewater Spotted Fever is that it's actually a less severe disease than Rocky Mountain Spotted Fever. It has a much better prognosis," Fisher said.

Crosley took antibiotics for two weeks and is now recuperating. Doctors believe there will be no long-term effects.
Fisher told 10 On Your Side the best way to protect yourself is to avoid a bite by using a repellent. If you do have a tick on your body, slowly remove it using a pair of tweezers.

maladie de lyme : attaques de panique

Lyme disease panic attacks

Off-the-Charts Anxiety: Is a Tick Bite Making You Nuts?

Tick-borne infections aren't just causing sore joints and swollen knees. Some cause psychiatric symptoms that often go untreated, some experts say. 

By Leah Zerber
 
 If you're not careful, this little guy could quite literally drive you crazy!
 
If you're not careful, this little guy could quite literally drive you crazy!
Finding an engorged, blood-sucking tick attached to your skin can cause anxiety in and of itself. After all, Lyme disease, an infection that causes multi-systemic, waxing-and-waning symptoms, and a disease that isn't always detected or effectively treated early on, is on the rise. But researchers are starting to realize that, although getting bitten may be stressful, tick-borne infections could actually trigger panic attacks and other psychiatric disorders in some people. "After treating thousands of patients with tick-borne disease in the past 20 years, it appears psychiatric symptoms are more commonly seen when there is a co-infection," explains psychiatrist Robert Bransfield, MD, former president of the International Lyme and Associated Diseases Society (ILADS) and president of the New Jersey Psychiatric Association. Co-infections (when a tick passes along more than one disease) most often involve Lyme, babesiosis, a malaria-like infection that can cause fever, night sweats, and anemia; and bartonella (cat scratch fever), a bacterial infection that causes fever, headache, and raised skin rashes. Co-infections are most often culprits in tick-related panic attacks and anxiety, and these multiple infections from tick bites are quite common, occurring in an estimated 30 percent of cases.
Dr. Bransfield, who is also associate director of psychiatry at Riverview Medical Center in New Jersey (a state with a high prevalence of Lyme disease), points out that 240 peer-reviewed scientific articles demonstrate an association between Lyme and other tick-borne diseases and mental illness. For instance, a small study published in The Clinical Journal of Pain in 2005 found that patients experiencing panic attacks also suffered other symptoms not typical of standard panic attacks—extreme sensitivity to light, touch, and sounds, joint pain, mental fogginess, and migrating pain, all of which can be symptoms of Lyme disease—and those people tested positive for Lyme and babesiosis, which, like Lyme, is on the rise in the U.S. Once treated with antibiotics for both diseases, the patients no longer experienced panic attacks.
Another study published in the American Journal of Psychiatry in 1994 found that 40 percent of patients with Lyme disease develop neurological impairment, which may not surface for months or years after a tick bite. Psychiatric reactions included not only panic attacks, but also bipolar disorder, schizophrenia, dementia, obsessive-compulsive disorder, anorexia, and depression.
"Many of the psychiatric symptoms of Lyme and associated tick-borne diseases are mediated by immune mechanisms," Dr. Bransfield explains, adding that the in Lyme sufferers, the immune system gets thrown out of whack. Furthermore, things like infection and stress can weaken and provoke the immune system, causing chronic inflammation, which has been linked to mental disorders. Ultimately, there needs to be better interaction between infectious disease specialists, immunologists, and mental health practitioners, Dr. Bransfield says.
Tick-borne diseases could, quite literally, push you over the psychological edge. And although other insect-borne diseases like West Nile virus may garner more headlines, you're far more likely to be sickened by a tick, according to U.S. Centers for Disease Control and Prevention statistics. For instance, 2009 saw 720 cases of West Nile virus, while nearly 40,000 probable cases of Lyme disease occurred that year. (Many doctors specializing in Lyme treatment believe Lyme disease numbers could be much higher because tests detecting the disease are not very reliable.)

Here's how to gauge whether your anxiety is linked to Lyme disease or other tick-borne diseases:
• Know how different panic attacks work. Panic attacks spurred by Lyme disease or other tick-borne infections are generally different than non-infectious-based panic attacks, explains Dr. Bransfield. A regular panic attack lasts a few minutes, but he says those brought on by tick-related ailments can go on for more than a half hour. If your panic attack symptoms grow worse while on once-effective antianxiety treatment, it's another sign that Lyme or a related infection could be causing the attacks.
• Know when to consider tick-borne diseases. Don't rely on finding a tick attached to your body to gauge your Lyme disease risk: Many people don’t recall being bitten at all, while others notice migrating rashes or red or black-and-blue splotches shortly after being bitten. Other early Lyme symptoms sometimes pop up a few days to a month after infection and include fatigue, fever, and chills. If the disease becomes more established in your body, it could cause cardiac and neurological problems. If you think you've been recently infected with Lyme, ask your doctor to perform blood tests, and if negative, have them repeated about six weeks later. If the results are still negative and you still suspect Lyme, you may want to see a doctor who specializes in treating Lyme aggressively. Doctors should first test to rule out other conditions with similar symptoms.
• Fight with your doctor if you need to. Lyme disease is a contentious subject, with two different schools of thought: Some consider to be a short-term infection, others believe it can be chronic. Some doctors take the threat of chronic Lyme seriously, and believe it should be treated with longer courses of antibiotics; others believe chronic Lyme doesn't exist. (Read Lyme Hearing Highlights a Broken System and Lyme Disease Treatment Guidelines: All Wrong? for more background.) Until more doctors recognize the severity of the disease, if you believe you have Lyme it's best to advocate for a clinical diagnosis using the strategy above.
Here are three steps to keep Lyme disease from driving you crazy in the first place:
• Scan daily. You slash your risk of developing Lyme disease if you scan your body daily for ticks. Use a mirror or a partner to check hard-to-see places, and pay special attention to areas where ticks like to hide—armpits, bra and panty lines, and the groin. And don't forget to keep your pet free of ticks naturally, too.
• Scrub-a-dub-dub. Bath time presents a great opportunity for Lyme prevention because of these three things:
1. It's another chance to focus on your body and check for ticks.
2. You can wash off ticks that haven't attached yet.
3. You may take off clothing ticks were hanging onto.
For the best protection, take a shower within two hours of being outside.
• Don't rely on a bull's eye or blood tests. While many people associate a bull's eye rash with Lyme disease, only about 30 percent of people who contract the infection show the telltale symptom. Blood tests are also unreliable, and many doctors specializing in Lyme treatment believe clinical diagnosis is more effective than blood tests alone. "The science surrounding testing and the interpretation of currently available tests is a complex subject," says Dr. Bransfield. "Negative tests may occur when infection is present; there is no currently available test that can reliably rule out the presence of tick-borne disease."
If your family doctor has ruled out other health issues and you still believe you have Lyme disease or another tick-borne disease, join Lyme support groups and ask for doctor recommendations.

bataille contre lyme



June 24, 2012

Local woman battles Lyme disease

CULLMAN — Imagine going for years and years with an undiagnosed disease that causes your joints to swell and ache, constant ear, upper respiratory and bladder infections, migraines, depression and eventually fibromyalgia.

No one knows what the root cause is, so you are only treated for the symptoms, never really getting well from one bout of infections before another takes its place.

Sometimes people think you are a hypochondriac, or that you suffer from some form of mental illness.

This has been 33-year-old Christy Driver’s life up to now. It wasn’t until the spring of 2010 that an astute doctor who was actually treating Christy’s oldest daughter for Aspergers Syndrome, decided to test her for Lyme disease, which is normally transmitted through the bite of an infected tick.

“I don’t actually remember being bitten by the tick that carried the disease, but when I was a child we lived along the Flint River, and there were woods all around our house where we played,” Christy recalls.

“My mother had been bitten by a tick and became very sick, so she was always vigilant about checking us for them, and found several over the years on us,” Christy recalls.

Christy, who worked as a paramedic on an ambulance, attended UAB, earning a certificate in Emergency Medicine. Her husband, Shane Driver, a 1991 graduate of Good Hope, is a paramedic for Air Evac.

The couple, who live in Good Hope, have two children, Erin, 11, and Allison, 7.

After Allison was born, Christy’s symptoms worsened. She received a Rubella booster in 2005, and right away began experiencing almost non-stop migraines. “I think that shot contained just enough to overload my immune system and bring on the symptoms,” she said.

The young mother also suffered from chronic fatigue and joint pain. Some doctors put these symptoms down to the “baby blues”, treating her with pain medication and anti-depressants, which masked the symptoms somewhat, but did nothing to dispel them.

“Over the years I went to seven or eight different doctors, trying to get an answer,” said Christy. “Some called it ‘drug fevers’ and others treated the depression and infections, but nothing really helped. A couple of them even told my husband that I was seeking attention,” she said ironically.

She was on a string of medications a “mile long” and doing her best to be a wife and a mother to two young girls, keeping house, and working up until the point where she realized that she couldn’t remember things and even forgot how to start her car, perhaps due to the prolonged inflammation around her brain.

“I remember being in the Wal-Mart parking lot and I just couldn’t remember what to do to start the car,” she said. “I called Shane crying and told him I couldn’t make the car work.”

Now she never ventures out alone.

Christy had been bedridden for almost two weeks when her daughter’s doctor agreed to see her the first time.

She and Shane traveled to Nashville.

After looking over her list of symptoms and reviewing her previous test results, the doctor looked at her and asked, “Have you ever been tested for Lyme Disease?”

When she answered “no”, he went on to explain to her that they would draw blood for a Western Blot Test, to be sent to the Igenex Lab.

“These are the symptoms,” he said, handing Shane a list of what turned out to be exactly what Christy had been experiencing.

The test came back “highly positive” for Lyme disease.

“When he started reading me the results I knew that this was finally a definitive diagnosis,” said Christy. “I went home and did more research, which just led me to the same conclusion.”

Shane came to the conclusion that, although Christy had been in and out of doctor's offices for constant childhood illnesses, no one had thought to link it to a chronic illness, such as Lyme disease.

“I was just glad to have a diagnosis,” said Shane. “Now I knew that she wasn’t mentally ill.”

“Modern medical doctors don’t consider this a ‘high-risk’ area so they don’t often test for it,” explained Christy. “For years I have had this little parasite inside me, wrecking havoc with my body, and it took that long for someone to recognize what it was.”

Christy’s research and her trips to the doctor have given her a wealth of knowledge in the way Lyme disease works.

“There are three types of antibodies, one starts out as a spirochete, then it goes to a second form, called a L-Formation, and then it goes into a third type called a cystic form,” she said.

She has all three forms, which are constantly changing, making it almost impossible for normal blood work to detect it.

“The best way to describe this process is that at any point it can go into remission, like cancer,” explained Shane, whose medical background allowed him to better understand what was happening to his wife. He and Christy have done hours and hours of research since the diagnosis was made.

“Normally, a person would exhibit flu-like symptoms anywhere from 48 hours to six weeks after being bitten by an infected tick,” he continued. “With Christy, it was undiagnosed for decades, so it has taken over her body. It has been a nightmare….” Shane said forcefully.

“I never had a bullseye rash that I could see,” said Christy. “That is one thing that doctors always look for, but it could have been under my hair and no one would have suspected that it was there.

“According to recent research, there is even reason to believe that the disease can be transmitted from the moment of conception if the antibodies are active at that precise time,” Christy said. Since her own mother was bitten by a tick before she was born, she is concerned that she might possibly have been born with Lyme Disease. These fears are compounded by the fact that her mother has exhibited some of the symptoms for years.


“Only about 10 percent of cases result in the bullseye rash,” she continued. “I’m really not a normal case because mine when undiagnosed for so long. Normally, a person would get sick and be treated with antibiotics in the first few days, or weeks, when it can be successfully cured. With mine, the treatments are ongoing and they really don’t know the prognosis at this point.”

In August of 2010, Christy had to quit her job because of her condition. She has gone from taking oral medications to having an intravenous tube called a PICC line inserted into a vein, delivering constant antibiotics to combat her worsening symptoms.

After about six months the PICC line became so inflamed that it had to be removed. Now she is waiting for a Hickman port to be implanted, but meanwhile she is taking large oral doses of the antibiotic Amoxicillin.

The Drivers now require help running the household and caring for their children. “My memory is not functioning at full capacity,” said Christy. “At one point I was writing notes to myself about even the simplest tasks.”

Shane described her behavior this way, “She could not process the simplest information, like ‘put the mail in the mailbox’ or other easy instructions. We were headed for divorce, financial ruin, or maybe having her committed to somewhere that she could get help.”

“This thing is so hard to diagnose,” he said. “If you google it, the information will tell you that there is low incidence of Lyme Disease in this area, but that’s because it’s misdiagnosed so often.”

The Drivers’ research has turned up information linking Lyme disease with both autism and Alzheimers disease. “I think there are a lot of people who are misdiagnosed with those conditions who are really suffering from Lyme Disease,” said Christy.

Another reason for possible misdiagnosis is that officially this disease doesn’t exist in this area. “It’s hard to get funding for something that doesn’t exist,” said Shane, with more than a little sarcasm.

There are few treatment options in cases like Christy’s. Most patients are treated with massive doses of antibiotics. One thing that seems to relieve Christy’s joint pain somewhat is being in a swimming pool. However, she won’t be able to get in the pool for a while because of her Hickman port, which can’t get wet until the site is healed.

Another controversial treatment, which claims to have has some success in causing various cancers to go into remission, called a Rife machine, works on the assumption that the antibodies can literally be shaken out of remission so that they can be attacked by the medication.

“When these antibodies develop a bio-film around themselves the medications just bounce off of them,” described Shane.

Because they can lie dormant in the cells of the body, they can “hide” from a normal blood test and if the antibody is not active, it can’t be killed. The Rife machine works on the hypothesis that if the antibodies are shaken into an active stage, they can then be zapped by the antibiotics.

The Rife machine is so new that the costs are prohibitive and insurance doesn’t cover it, of course, so there is a fund set up for the purchase of the machine in hopes that it will be the answer.

"Awareness needs to be made in this area since so few know anything about Lyme Disease," said Christy. "If this article helps anyone find some answers to their health problems, then that will be more than I can ever ask for."



Lyme Disease; Most Common Chronic Neurologic Signs and  Symptoms



Memory Loss 81%

Fatigue 74%

Headache 48%

Sensory loss 44%

Spinal or radicular pain 41%

Depression 37%

Sleep disturbances 30%

Distal Paresthesis 26%

Irritability 26%

Difficulty in finding words  19%

Fibromyalgia 15%

Hearing loss 15%



RF Kaplan and AC Steere Chronic neurologic manifestations of Lyme disease New England Journal of Medicine 1990  323121} 1438



Fast Facts;

Lyme Disease; Adult Symptoms



Lyme is the fastest growing vector-borne disease

85% do not recall the tick bite

Less than 70% of people develop a rash

Lab tests may be negative in the first 4-6 weeks



Early symptoms:

Flu-like symptoms (fever, chills, sweats, muscles aches, fatigue, nausea and joint pain)

Rash (10% have EM rash)

Bell's Palsey



Later Symptoms:

Headache

Stiff neck

Light or sound sensitivity

Cognitive impairment

Sleep Disturbance

Depression, anxiety, or mood swings

Arthritis

Fatigue

Abdominal pain, nausea, diarrhea

Chest pain, palpitations

Shortness of breath

Tingling, burning or shooting pains



Sidebar

If you would like to learn more about Lyme Disease, visit www.underourskin.com to view an informative documentary about the disease.

If you would like to donate to the Driver family to help defray the costs of medications and treatments, visit www.gofundme.com/Christy

les biotoxines


The Biotoxin Pathway

Stage 1: Biotoxin Effects

It all starts when a person is exposed to a biotoxin. In most people, the biotoxin is 'tagged' and identified by the body's immune system and is broken down and removed from the blood by the liver. However, some individuals do not have the immune response genes (HLA-DR genes) that are required to eventually form an antibody to a given foreign antigen. In these cases the biotoxins are not 'tagged' and remain in the body indefinitely, free to circulate and wreak havoc.
Once present in the body, the biotoxins begin to set off a complex cascade of biochemical events. The biotoxin binds to surface receptors (Toll receptors and many more) in nearly every kind of cell in the body. This recognition and binding of the biotoxin causes a continual upregulation of multiple inflammatory pathways, including production of cytokines, split product of complement, and TGF Beta-1. Biotoxins also directly affect nerve cell function, which is one of the reasons that the symptoms and visual contrast sensitivity (VCS) test are so useful in diagnosis.

Stage 2: Cytokine Effects

Cytokines in turn bind to their receptors, causing release of MMP9 in blood.  In the brain, cytokines bind to the leptin receptor, preventing its normal function in the hypothalamus.  The blocked leptin receptor will no longer create the initiation of steps that lead to production of alpha melanocyte stimulating hormone (MSH).  Elevated cytokines can produce many different symptoms including: headache, muscle ache, unstable temperature and difficulty concentrating.  This problem is the disastrous effect of MSH deficiency.  High levels of cytokines can also result in increased levels of important compounds such as I-1 and clotting factors as shown by a von Willebrand’s profile. Of importance in cardio vascular health, MMP-9 delivers inflammatory elements from the blood into sensitive tissues and can combine with PAI-1 to increase clot formation and arterial blockage.

Stage 3: Reduced VEGF

The elevated cytokine levels in the capillaries attract white blood cells, leading to restricted blood flow and lower oxygen levels in the tissues (we call this capillary hypoperfusion). Reduced VEGF leads to fatigue, muscle cramps and shortness of breath.

Stage 4: Immune System Effects

Patients with certain HLA genotypes (immunity related genes) may develop inappropriate immune responses which may include antibodies to:  gliadin (gluten sensitivity), actin, anca (think ulcerative colitis), cardiolipins (affects blood clotting), and more. Most devastatingly of all, the complement system becomes chronically activated resulting in high levels of C4a.

Stage 5: Low MSH

Reduced MSH production results in yet another set of problems and symptoms. The production of melatonin is reduced which results in sleep problems. Endorphin production is suppressed which leads to chronic and sometimes unusual pain. Lack of MSH can cause malabsorption or 'leaky gut' which further weakens and deregulates the immune system. White blood cells eventually lose regulation of cytokine response so that opportunistic infections may occur or recovery from infections is slower.

Stage 6: Antibiotic Resistant Staph Bacteria

Reduced MSH also allows resistant staph (MARCoNS) to survive in biofilm on the mucous membranes. These bacteria further compound MSH deficiency and the problem by producing exotoxins A and B that cleave MSH, further decreasing the MSH levels. At this point, the downward spiral starts to perpetuate itself.

Stage 6: Pituitary Hormone Effects

Reduced MSH can decrease pituitary production of antidiuretic hormone (ADH) which can lead to thirst, frequent urination, neurally-mediated hypotension (NMH), low blood volume, and electric shocks from static electricity. While sex hormone production is often down-regulated the pituitary may upregulate the production of cortisol and ACTH in the early stages of illness, then drop to abnormally low, or low-normal ranges later.
Click Here to Download the Biotoxin Pathway


Many folks with Lyme find they are highly sensitive to mold : les lymés sont très sensibles aux moisissures


http://www.survivingmold.com/diagnosis/molds-mycotoxins-more

Molds, Mycotoxins, & More

Many health effects are caused by exposure to the interior environment of Water Damaged Buildings (WDB).  The complex mixture of contaminents present in the air and in the dust in WDB form a toxic chemical stew.
There are so many possible sources of these toxic compounds found in WDB that can lead to the variety of symptoms caused by illness, no single compound can be identified as the sole cause of the inflammatory responses, or the illness, seen in affected patients.  Since no one thing can be deemed as solely responsible for the sickness, the sole cause becomes the WDB itself.
Below is a list of of some of these dangerous compounds and an explanation of each.  Please understand this toxic chemical stew is a very complex mixture that truly wreaks havoc in the body.  These explanations are simplified to make them easier to understand.
Knowledge is power and patients have the right to understand what is making them sick, so they can avoid exposure in the future.
  • Fungi (fun´ji) - A single-celled or multicellular organism. Fungi can be true pathogens that cause infections in healthy persons or they can be opportunistic pathogens that cause infections in immunocompromised persons.
     
  • Bacteria (bak-tēr´e-ah) - Single-celled microorganisms which can exist either as independent (free-living) organisms or as parasites (dependent upon another organism for life).
     
  • Actinomycetes - A group of gram-positive bacteria (order Actinomycetales) that produce various bioactive agents.
     
  • Mycobacteria [mī′kōbaktir′ē·ə] - A large family of bacteria that have unusually waxy cell walls that are resistant to digestion.
     
  • Mold (mōld) - Mold refers to multiple types of fungi that grow in filaments and reproduce by forming spores. Mold may grow indoors or outdoors and thrives in damp, warm, and humid environments. Mold can be found in essentially any environment or season.
    The most common types of mold that are found indoors include Cladosporium, Penicillium, Alternaria, and Aspergillus. Stachybotrys chartarum (also known as Stachybotrys atra and sometimes referred to as "black mold") is a greenish-black mold that can also be found indoors. Stachybotrys grows on household surfaces that have high cellulose content, such as wood, fiberboard, gypsum board, paper, dust, and lint.
    Molds reproduce by forming tiny spores that not visible to the naked eye. Mold spores are very hardy and can survive under conditions in which mold cannot grow, such as in dry and harsh environments. These spores travel through outdoor and indoor air. When mold spores land on a surface where moisture is present, mold can start to grow.
  • Spore (spor) - tiny spores that are not visible to the naked eye produced by mold. Mold spores are very hardy and can survive under conditions in which mold cannot grow, such as in dry and harsh environments. These spores travel through outdoor and indoor air. When mold spores land on a surface where moisture is present, mold can start to grow.

  • Mycotoxins (mī′kōtok′sin)  - toxic chemicals that are present on spores and small fragments of mold and fungus that are released into the air.

  • Endotoxins (en′dōtok′sin) - also called Lipopolysaccharides (LPS), are cell wall components of Gram negative bacteria.  They are shed into the environment of WDB upon death of the bacteria.  LPS cause inflammatory responses via signaling pathways in the body, releasing inflammatory cytokines.  LPS aggravate existing lung disease (asthma, HP), can cause inflammation of the lungs and are synergistic with mycotoxins.

  • Inflammagens (in-flam´ah-jen) - an irritant that elicits both edema and the cellular response of inflammation

  • Beta Glucans (beta-glucans) - are polysaccharides of D-glucose monomers linked by β-glycosidic bonds. β-glucans are a diverse group of molecules that can vary with respect to molecular mass, solubility, viscosity, and three-dimensional configuration.

  • Hemolysins - exotoxins produced by bacteria that cause lysis of red blood cells in vitro.

  • Microbial Volatile Organic Compounds (VOCs) - Microbes can release organic compounds into the air when there is adequate food supply for such “secondary metabolite” production.  These volatile compounds, called mVOCs for short, can give basements their distinctive musty odor as well as activate innate immune responses in susceptible patients.  While we think of fungi as the most common producers of mVOCs, bacteria and actinomycetes are indoor-producers as well.

des fragments de bactéries restent dans les articulations apres traitement , les antigènes des spirochètesne partent pas .

 Recent study claims that "scientists discovered bacterial fragments in the joints of mice treated for Lyme disease that may be the underlying cause of persistent arthritis-like joint pain following Lyme infections..."

 

Fragments of Lyme Disease Bacteria Linger in Joints After Treatment


Scientists discovered bacterial fragments in the joints of mice treated for Lyme disease that may be the underlying cause of persistent arthritis-like joint pain following Lyme infections, says a report in the Journal of Clinical Investigation.
Lyme disease is caused by Borrelia burgdorferi, a worm-shaped bacterium also called a spirochete that is transmitted by tick bites. There were more than 22,500 confirmed cases of Lyme disease and another 7,600 probable cases in the U.S. in 2010, according to the Centers for Disease Control and Prevention. Infections usually start with a skin rash but can damage the heart, joints, and nervous system. Lingering joint pain affects up to 25% of people, despite effective treatment with antibiotics.
Yale University researchers induced Lyme infections in mice with impaired immune systems and control mice. Both groups received either antibiotic-treated drinking water sweetened with sugar or sweetened water only for 30 days. The antibiotics killed Lyme bacteria in control mice and all but one of the immune-compromised mice; Lyme bacteria persisted in untreated mice.
Researchers found remnants of Lyme spirochetes in the knee joints of all antibiotic-treated mice, indicating the antibiotics eliminated the bacteria but not the residual debris. Spirochetes were found adjacent to ear cartilage in most of the mice. These bacterial deposits were capable of triggering an inflammatory response but not a full-blown Lyme infection, the study showed.
Caveat: The research hasn't been tested in humans.
—Ann Lukits Title: Spirochete antigens persist near cartilage after murine Lyme borreliosis therapy
A version of this article appeared July 31, 2012, on page D4 in the U.S. edition of The Wall Street Journal, with the headline: After Lyme Disease Treatment, Bacterial Debris Lingers in Joints.

Scientist Draws Possible Connection Between Morgellons Disease And GMOs – Audio


temoignage :
La maladie du morgellon… voila plus de 7 ans que mon épouse en souffre.
En voici les symptômes tels que nous le vivons :
Ce qui sort de la peau : des grains noirs ( en abondance parfois), des fils noirs ( parfois animés), des petites choses dures indéfinissables ( genre bouts de bois , de paille, des petites pierres de couleurs ou translucide) des parties d’insectes ( moustiques , araignées, des vers) des insectes vivants et volants ne sont pas à exclure.
En outre : Des douleurs liées à des “bosses”, des chatouillements intenses, des lésions spontanées avec parfois des saignements ( visage, bras , jambes , dos )
Une fatigue permanente, de la confusion mentale, des troubles psychiques ( troubles de bipolarités, dépression, poussées suicidaires)
Nous sommes dans un enfer car notre situation c’est totalement dégradée.

  George Schwartz de Santa-Fé, Nouveau Mexique qui a tout d’abord pensé que la bactérie Stenotrophomonas maltophilia était la cause de la maladie, a déclaré avoir traité avec succès des patients avec des antibiotiques qui ciblaient cette bactérie hydrophile15. Il pense maintenant que cette infection est d’origine parasitaire. Dans cet ouvrage Lisa's Disease, A Fiber Disease (« La maladie de Lisa, une maladie fibreuse »), il décrit quatre stades dans cette affection (le stade quatre relevant de la supercherie). Schwartz traite aussi ses patients avec une médication anti-vermineuse et de la terre de diatomées18. Il déclare « C’est une peste des temps modernes qui croit silencieusement chez l’hôte pendant des semaines pour exploser en lésions cutanées allant jusqu’à des lésions caverneuses, avec production d’œufs et de larves. C’est une affection hautement contagieuse, une pandémie qui atteindra bientôt une masse critique. » Il a aussi développé un plan de traitement pour les premiers stades19. En août, 2006 une nouvelle brochure vient d’être publiée ; elle décrit la maladie avec des plans de traitement20

 

 

From CBS radio in Tampa. Possible connection between genetically modified organisms and Morgellons? (Some folks who have Morgellons--a mysterious skin ailment--also have Lyme disease.)

 

Scientist Draws Possible Connection Between Morgellons Disease And GMOs – Audio

Elizabeth Dougherty, Special correspondent, Food Nation Radio Network

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(Photo JORGE DIRKX/AFP/Getty Images)
(Photo JORGE DIRKX/AFP/Getty Images)
headshot3 From Food Nation Radio
For about two years now, Food Nation Radio Network has been covering the issues that affect our food supply. During the course of our research, investigations and interviews for the show, we came across a particularly disturbing piece of information in the genetically modified food puzzle. It’s the possible relationship between agrobacterium, genetic engineering and Morgellons Disease.
Agrobacterium is a bacteria that causes tumors in plants through a transfer of DNA. It is used for genetic engineering of corn, soybeans, canola, sugar beets, alfalfa and other foodstuffs. Some studies have shown agrobacterium can also affect the DNA of humans.
A study done on Morgellons Disease patients by Vitaly Citovsky, a professor of molecular and cell biology at Stony Brook University in New York (SUNY) found all patients tested positive for the presence of agrobacterium, while the healthy control patients did not. Morgellons is a disease one would think would be in a science fiction novel. It is characterized by lesions on patients and fibers containing minerals growing underneath the skin. For many years, nearly the entire medical community (including the CDC) maintained it was a psychiatric condition, with patients causing their own lesions and other symptoms. As of January, 2012 the CDC appears to maintain that stance, although researchers and respected scientists in Oklahoma, New York, Toronto and other parts of the world are taking this possible epidemic seriously.
Some notable individuals claim to suffer from Morgellons, including musician, Joni Mitchell and former baseball player Billy Koch. It is possible this is an infectious disease, due to the presence of it in entire families and it is found more among nurses and teachers who come into contact with a number of people on a daily basis. Morgellons is reported more in California, Florida and Texas than anywhere else, although it is found throughout the world.
Here is our recent interview with accomplished genetic researcher, Professor Joe Cummins, Professor Emeritus at the University of Western Ontario on the possible Morgellons Disease/GMO connection FNRN Highlight – Prof Joe Cummins on Morgellons .

Elizabeth Dougherty has been a food writer for over 10 years, attended culinary school and holds a Bachelor’s degree, Magna Cum Laude in Hospitality, Business and Labor Relations from NYIT. She has been a talk show host of nearly 150 episodes of Food Nation Radio which airs each Saturday morning at 6 on AM1010 CBS and other stations. You can read her articles and hear previous shows on her podcast page on the Food Nation Radio Network website and on Facebook.

infoclimat , échanges sur lyme

http://forums.infoclimat.fr/topic/80072-gare-aux-tiques-et-a-la-maladie-de-lyme/

doctors recognize that Lyme can be long-term

http://video.foxnews.com/v/1759740368001/how-to-protect-yourself-against-lyme-disease/

video à regarder

 
 
Pretty good segment about Lyme on Fox news. The doctors recognize that Lyme can be long-term, that it can affect the brain profoundly, that not everybody gets a bull's-eye rash, and mentions IV antibiotics as potentially helpful.

la maladie de lyme est dangereuse mais on peut en guérir...mais il faut des années

Lyme disease is dangerous but curable

Seacoast cases have swelled over the past 10 years
A female deer tick.Courtesy photo
Lyme disease may be one of the most frightening, yet misunderstood diseases. It is often missed or misdiagnosed, sometimes for so long the effects can be devastating.
Lyme disease is contracted from the bite of an infected deer tick.

At a glance

Avoiding Tick Bites
Stay on trails outdoors: Avoid areas of overgrown brush and tall grasses. Wear light-colored clothes so ticks can be easily seen. Wear a hat, long-sleeved shirt and long pants tucked into boots or socks. Check yourself often for ticks. Use insect repellent containing DEET or permethrin (follow directions).
Removing an Attached Tick
Remove the tick promptly: The sooner you remove it, the less chance of infection. Use tweezers to grasp the tick's mouthparts at the surface of the skin. With a steady motion, gently pull the tick straight out. Wipe the bite area with an antiseptic, or wash with soap and water. Be alert for symptoms of illness over the next 7-10 days. Do not squeeze the tick. Do not rub petroleum jelly on the tick. Do not use a hot match or cigarette. Do not pour kerosene or nail polish on it. Wash immediately with soap and water, and if you suspect Lyme, save the tick in a jar to bring to the doctor's office.
"We have had a rapid increase of Lyme in the past 10 years," said Dr. Jodie Dionne-Odom, N.H. deputy state epidemiologist. "It peaked in 2008, at 1,300 cases. Currently, it is down to about 1,200 yearly reported cases. This only represents the documented cases. Some people are never reported."
The rate of N.H. cases is similar to other New England states. Dionne-Odom said cases are moving northward, with the greatest prevalence now being in the Seacoast.
"We do educational seminars for physicians and the community," she said. "Most primary-care doctors and pediatricians are well versed in the disease and its symptoms."
The trademark bull's-eye rash is usually present, but not always, and finding the infected tick right away makes the difference. Dionne-Odom said it takes 24 to 36 hours for the tick to regurgitate the contents of its stomach into the blood stream and that's where the bacteria live. Caught early, a short course of antibiotics is usually effective. She stressed Lyme disease is curable. Even if it crosses the blood brain barrier, IV antibiotic therapy can be used effectively.
"The problem is that many people do not recognize what they have, may not get the rash or see the tick," she said.
Ticks tend to attach to warm, moist areas, like the back of the knee, neck or groin.
"Undetected, the bacteria will cause organ or joint damage and those can be hard to repair," Dionne-Odom said. "Some people go years before diagnosed. Lyme encephalopathy, that cognitive damage to the brain, requires further testing to determine damage."
Katherine Lagassie, 18, of Maine, is struggling with college because of her Lyme disease. She was diagnosed as a high school freshman but she and her family, who all have Lyme disease, believe she contracted it at the age of 8.
"When I got sick, they at first dismissed Lyme because I never got the bull's-eye rash," she said. "I started going to doctors at the age of 10. I was always sick."
Lagassie was chronically exhausted. Her muscles ached so badly she could no longer ride her bike or swim in the family pool.
"At 10 years old, my back hurt so badly, I would have to lie on the floor," Lagassie said. "I was tested for everything — arthritis, even MS. I spent months at home, not being able to go to school. I just stayed in bed."
Finally, Lagassie said, in a last-ditch effort, a Kennebunk doctor tested her for Lyme disease. It was positive. Lagassie was treated with antibiotics and naturopath medications, 32 pills a day. Eventually, her diagnosis included a secondary infection, babesiosis, requiring a second type of treatment. She still takes myriad medications, designed to keep her immune system strong.
"If I crash, I don't come back easily," she said. "When I started college, I was pre-med. I had to adjust that because I could not handle the work load. I still plan to be in health care, but have lowered my sights to a less strenuous course of study. I had to arrange for extended test times, because of my cognitive difficulties. I sometimes get so anxious and paranoid, I can't recover information I know I have inside."
Brian Lagassie, Katherine's father, said that since the medical community often does not recognize chronic Lyme disease, insurance plans will not cover many treatments.
"At points, her treatments were costing $1,400 a month," he said. "Disability laws refuse her aid, because while she can't work, they refuse to acknowledge it. Face to face, you'd never know how devastating this is for her. Lyme disease patients are much where AIDS patients used to be. They are stuck in between, and the medical community needs to recognize the human side."
A common sign of Lyme disease is a red bull's-eye rash expanding rash 2 inches or more in diameter, which appears between three days and a month after the tick bite. People with Lyme disease might get chills and fever, headaches, or muscle and joint pain, and often feel tired. Early symptoms resemble the flu. They can come and go. Many people don't get the rash and may have false negative tests.
There are three stages of the disease. Stage 1 is localized, meaning the disease has not yet spread to the blood stream and the body. In stages 2 and 3, there are varying degrees of dissemination through the blood stream, with accompanying joints, muscles and organs affected.
If Lyme disease isn't treated properly, other signs can appear weeks or months after the tick bite. These include arthritis, numbness or paralysis, often in the face muscles, problems with heart rhythm and problems with memory or concentration.
The blood test for Lyme disease is called ELISA. If it is positive, a western blot test can confirm the diagnosis. A regimen of high doses of antibiotics is generally begun within 72 hours of the discovery of the tick.
Complicating diagnosis is that ticks can transmit other diseases simultaneously, including anaplasmosis and babesiosis. Symptoms can be similar, but both require different courses of treatment. If not recognized as parallel infections, treatment will be ineffective.
Dr. David Itkin is an infectious disease specialist and the epidemiologist for Portsmouth Regional Hospital. He said there is no scientific basis to support long-term symptoms people claim as being part of their Lyme disease.
"More I would say it's the way disease affects the person," he said. "For some, their immune system takes longer to recover. The symptoms are similar to chronic fatigue syndrome, and appear to be the after-effects of the disease, as the immune system rebuilds."
Itkin said there is also no documented evidence to say Lyme causes cognitive defects. The question is how to treat those patients who do seem to be suffering from an undefinable ailment.
"We now have a greater recognition of this disease as a moving target," he said. "Fifteen years ago, it was not here. It was in Ipswich, Cape Cod, but in the last decade it has been moving northward."
Testing for Lyme disease is not done routinely. Itkin said he'd be more likely to test based on presentation.
"A landscaper with the suspect symptoms is more likely to be tested than an 89-year-old man who lives in a nursing home and never goes outdoors," Itkin said. "Lyme does not present as respiratory problems, or with diarrhea. We need to rule out the obvious and test what fits as suspicious for the disease."
Some cases are more serious, requiring IV antibiotic therapy. Itkin said a Seacoast hospital recently treated a case of Lyme carditis, meaning the patient's heart had become involved.
Otherwise, a 14-28 day course of antibiotics is what he would prescribe for those with general symptoms such as Lyme arthritis.
David Hunter heads the Greater Manchester Lyme Disease Support Group. He said the group, started about four years ago, has more than 500 members from southern New Hampshire and Massachusetts.
"Twelve years ago, my daughter was diagnosed," Hunter said. "We lived in Connecticut at the time and she was in the eighth grade. When she was diagnosed, I thought it would be easy. Then she proceeded to miss high school. She was a number one student, soccer player, cello player, and she lost it all. So, I got involved. I was looking for answers and I found controversy. People in school, who should have supported her, said she had 'school phobia.' Suddenly her friends thought she was weird and they abandoned her."
Hunter's daughter is now 26. She still suffers symptoms but succeeded in getting her GED and graduating from the University of New Hampshire with high honors.
Hunter continues the support group, gives talks whenever asked and recently started a teen support group.
"Our meetings are always well attended," he said. "There are always people wanting to share their stories and get information. My mission is to make people understand."
To learn more about the support group, call Hunter at 660-3425.
At a glance
Avoiding Tick Bites
Stay on trails outdoors: Avoid areas of overgrown brush and tall grasses. Wear light-colored clothes so ticks can be easily seen. Wear a hat, long-sleeved shirt and long pants tucked into boots or socks. Check yourself often for ticks. Use insect repellent containing DEET or permethrin (follow directions).
Removing an Attached Tick
Remove the tick promptly: The sooner you remove it, the less chance of infection. Use tweezers to grasp the tick's mouthparts at the surface of the skin. With a steady motion, gently pull the tick straight out. Wipe the bite area with an antiseptic, or wash with soap and water. Be alert for symptoms of illness over the next 7-10 days. Do not squeeze the tick. Do not rub petroleum jelly on the tick. Do not use a hot match or cigarette. Do not pour kerosene or nail polish on it. Wash immediately with soap and water, and if you suspect Lyme, save the tick in a jar to bring to the doctor's office.

Tout ce que vous vouliez savoir sur les CD57


http://www.researchednutritionals.com/information.cfm?ID=200
 

Everything You Wanted to Know About CD57

By: GINGER SAVELY, RN, FNP-C From coast to coast, frustrations abound among patients and clinicians regarding the diagnosis of chronic Lyme disease. Misinformed health care providers in Texas and surrounding states consider the infection rare and non-endemic. They are inclined to rule out Lyme disease based on the negative result of a laboratory test that, unbeknownst to them, is highly insensitive. In the absence of a reliable laboratory test or adequate experience in the recognition of the varied and complex presentations of the illness, most clinicians are ill-equipped to diagnose chronic Lyme disease. Many patients suffer needlessly for years, hopelessly lost in the maze of the health care system, looking for answers and enduring the skepticism of practitioners inexperienced with the disease’s signs and symptoms.
What is needed is a better Lyme test or some other objective measure to persuade the practitioner to consider the diagnosis of chronic Lyme disease. Enter the CD57 test! You may have heard the term “CD57? tossed around on chat groups, or your Lyme-literate health care provider may have even explained the test to you in one of your moments of brain-fogged stupor. What is this number that sounds more like a type of Heinz steak sauce than a lab test, and what in the world does it have to do with Lyme disease?
Let’s start by going back to basic high school biology. You may remember that white blood cells (a.k.a. leukocytes) are the components of blood that help the body fight infections and other diseases. White blood cells can be categorized as either granulocytes or mononuclear leukocytes. Mononuclear leukocytes are further sub-grouped into monocytes and lymphocytes.
Lymphocytes, found in the blood, tissues and lymphoid organs, attack antigens (foreign proteins) in different ways. The main lymphocyte sub-types are B-cells, T-cells and natural killer (NK) cells. B-cells make antibodies that are stimulated by infection or vaccination. T-cells and NK cells, on the other hand, are the cellular aggressors in the immune system and are our main focus in the discussion that follows.
Let’s pause a moment and introduce something you probably never learned about in high school biology class: CD markers. CD, which stands for “cluster designation”, is a glycoprotein molecule on the cell surface that acts as an identifying marker. Think of comparing cells as comparing people. Humans are made up of innumerable superficial identifying characteristics (such as hair color, eye color, etc.) and so are cells. Cells probably have thousands of different identifying markers, or CDs, expressed on their surfaces, but 200 or so have been recognized and named so far.
Each different marker (or CD) on a cell is named with a number, which signifies nothing more than the order in which the CD was discovered. On any given cell there are many different cluster designation markers (CDs), giving each cell its unique appearance and function but also linking certain cells by their similarities (like grouping all people with brown hair or all people with blue eyes). Cells that have a certain kind of CD present on their surface are denoted as + for that CD type (e.g., a cell with CD57 markers on its surface is CD57+).
NK cells have their own specific surface markers. The predominant marker is CD56. The percentage of CD56+ NK cells is often measured in patients with chronic diseases as a marker of immune status: the lower the CD56 level, the weaker the immune system. You may have heard Chronic Fatigue Syndrome patients talk about their CD56 counts. A smaller population of NK cells are CD57+.
A below-normal count has been associated with chronic Lyme disease by the work of Drs. Raphael Stricker and Edward Winger. No one knows for sure why CD57+ NK cells are low in Lyme disease patients, but it is important to note that many disease states that are often confused with chronic Lyme (MS, systemic lupus, rheumatoid arthritis) are not associated with low CD57+ NK counts. The good news is that for most Lyme patients the CD57+ NK level increases as treatment progresses and health is regained.
CD57 markers can also be expressed on other kinds of cells, including T-cells, so it is important to distinguish between CD57+ T-cells and CD57+ NK cells. Clinicians need to be aware that many testing laboratories claiming to perform the CD57 test are actually looking at CD57+ T-cells rather than CD57+ NK cells, which are the cells of interest in chronic Lyme disease.
In order for a testing laboratory to measure the CD57+ NK level, it first measures the percentage of lymphocytes that are CD57+ NK cells. Then an absolute count is calculated by multiplying that percentage by the patient’s total lymphocyte count. The standard normal range for the absolute CD57 NK count is 60 to 360 cells per microliter of blood. This wide range was established based upon test results of hundreds of healthy patients. By these laboratory standards, a test result below 60 cells per microliter would be considered below normal and therefore associated with chronic Lyme disease. However, a recent study of my Austin patients has led me to believe that 100 cells per microliter is a more reliable threshold separating Lyme patients and healthy controls.
When Drs Stricker and Winger discovered that CD57+ NK cells are low in chronic Lyme patients and tend to increase with patients’ clinical improvement, an opportunity arose for Lyme-literate practitioners to utilize a handy tool to aid in the diagnosis of chronic Lyme disease, to follow treatment progress, and to determine treatment endpoint. Just as AIDS patients have always held great store in their CD4 T-cell count, Lyme patients now have a fairly reliable marker of the status of their illness.
It is important to remember that the CD57 result is just a number; far more important is the patient’s clinical status. An old professor of mine used to say, “treat the patient, not the lab test!” There is still much we do not know about the CD57 marker and what other factors may lower or raise it. However, overall, the CD57+ NK count is a useful tool in diagnosing and treating chronic Lyme disease in most patients. As a measure of immune status, it provides an indirect measure of bacterial load and severity of illness. Furthermore, in a patient who has a negative or indeterminate Lyme test but is highly suspect for the disease, the clinician may utilize the CD57+ NK count as one more piece in the complex puzzle of a Lyme disease diagnosis.
Postscript: If you would like your health care provider to order the CD57 NK test for you, your blood sample needs to be drawn into an EDTA tube (lavender top) on Monday through Thursday and sent immediately to either LabCorp in Burlington, NC, or Clinical Pathology Laboratories (CPL) in Austin, TX. LabCorp and CPL are the only two labs that perform this test properly. Quest does NOT. The LabCorp test code is #505026 and is named HNK1 (CD57) Panel. The CPL test code is #4886, CD57 for Lyme disease. The test is time-sensitive and must be performed within 12 hours of collection, so blood should not be drawn on a Friday or results may be inaccurate.
 

Lyme can be deadly serious

Message from Jenny O'Dea of Tick Talk Ireland
Jenny O'Dea 29 July 00:53
I know it's sad to post these things but I want any media or politicians know who happen to be passing that Lyme can be deadly serious - we MUST do more. During an interview on camera for the upcoming film I was told that Lyme is normally not that serious, I then relayed the death of my friend just before Xmas. Tears came to my eyes. She had ALS but also was positive for Lyme disease & thanks to a GMC hearing her Lyme treating doc could practise no more, no-one else would treat her, she lost all ability to speak or even swallow, she had a speaking machine & a hoist to get her in the bath. She passed away Dec last year...she had teenage children. OK this IS the worst case scenario but people I know of have been blinded & paralysed. A doc who came to the conference in Dublin was blinded through Lyme Disease which thankfully his sight returned on proper treatment, he now treats others (not here in Ireland sadly). Another doc in the States was diagnosed with ALS (Motor Neurone Disease) & given 6 months to live, he was treated for Lyme & recovered. We cannot idly sit by & let this happen. We're trying very hard to organise a meeting with government officials, I'll keep you posted! Meanwhile for crossovers between Lyme & ALS, MS, ME & more check out our links page at:
 http://ticktalkireland.wordpress.com/lyme-links/

Conférence du Dr Burrascano à Phoenix le 14 septembre 2012

EVENT: See Dr. Joe Burrascano Sept 14 in Phoenix

29th July 2012

burrascano for azlda






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Sponsored by the Arizona Lyme Disease Association.
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Top Lyme expert Dr. Joseph Burrascano will give a public lecture about tick-borne diseases in Phoenix, Arizona, on Friday, Sept. 14. Then, on Saturday and Sunday, he’ll address healthcare professionals at a CME-approved medical conference.
The following information is provided by the Arizona Lyme Disease Association:
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Joe Burrascano, Jr. MD
CLINICAL UPDATE ON TICK-BORNE DISEASES
Friday, September 14, 2012
6:30-7:45 pm
For both healthcare professionals and those interested in learning more about Lyme disease
$10 registration fee
Cash or check, payable at the event. Registration fee supports AzLDA outreach efforts.
Sheraton Crescent Hotel
2620 W. Dunlap Avenue
Phoenix, AZ
Dr. Burrascano is an internationally  recognized specialist in the diagnosis and treatment of Lyme and associated complex infectious diseases, and the chronic illnesses that accompany them. With over 25 years of experience and research in this field, he has appeared in and on virtually every form of media both here and abroad. He has also advised the CDC and NIH, testified before the U.S. Senate, an armed services joint subcommittee, and at various governor’s councils.
He is a founding member of the International Lyme and Associated Diseases Society, and is currently an active Board Member of the affiliated International Lyme and Associated Diseases Educational Foundation.
No longer in clinical practice, Dr. Burrascano works full time in the biotech arena to further medical research in tick-borne illnesses.
Notable among many of his ongoing projects  is his active involvement in the recent development of in vitro culturing methods of symbiotic Borrelia, including the Borrelia responsible for Lyme Disease.
Dr. Burrascano is still recruiting practitioners for his ongoing project, The Lyme and Associated Diseases Registry™ which follows each selected patient from the beginning to the end of their illness, including tracking their symptoms, tests, treatments and outcomes.
Also of interest
Dr. Burrascano and other well known faculty will speak at the CME approved conference DECODING THE MYSTERY OF CHRONIC ILLNESS to be held at the Sheraton Crescent on September 15-16.  Registration is open to medical practitioners and is designed to provide new skills and protocols to assist in the diagnosis and treatment of Lyme disease, Fibromyalgia and Chronic Fatigue Syndrome.  Full details are available at www.Decoding-The-Mystery.com